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Enhanced IL-17 signalling following myocardial ischaemia/reperfusion injury  

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File information File dimensions File size Options

Original JPG File

2956 × 1968 pixels (5.82 MP)

5.9 in × 3.9 in @ 500 PPI

 469 KB Restricted

Low resolution print

2000 × 1332 pixels (2.66 MP)

16.9 cm × 11.3 cm @ 300 PPI

 248 KB Restricted

Screen

1201 × 800 pixels (0.96 MP)

10.2 cm × 6.8 cm @ 300 PPI

 118 KB Restricted

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Resource details

Resource ID

275

Default
Publication
Retraction
Retraction Case 1
Retraction Case 2

Date

26 June 2019

Authors

Seán P. Barry, Samir Ounzain, James McCormick, Tiziano M. Scarabelli, Carol Chen-Scarabelli, Louis I.I. Saravolatz, Giuseppe Faggian, Alessandro Mazzucco, Hisanori Suzuki, Christoph Thiemermann, Richard A. Knight, David S. Latchman, Anastasis Stephanou

Publisher

Elsevier

Journal

International Journal of Cardiology

Subjects

BLS - Basic Life Sciences/Biochemistry,BLS - Basic Life Sciences/Biology - Cellular

Publication Date

24 October 2011

DOI

10.1016/j.ijcard.2011.08.849

Manipulation status

Retraction

Retraction DOI

10.1016/j.ijcard.2011.08.849

Related links

https://pubpeer.com/publications/C08DD8360123387DFEBBEFA3028898

http://retractionwatch.com/?s=david+latchman

Manipulated Image

5B

Manipiulation category 1

unclear

Comment 1

"The images in Figure 5B labelled Control, I/R and I/R+IL-17n both used a section of the same image, with additional features (yellow TUNEL labelled nuclei representing dying cells) having been either added or taken away;"
(See Retraction Notice)

Manipulated Image

5B

Manipiulation category 2

unclear

Comment 2

"In addition, the Control panel in Figure 5B appeared to be a horizontal flip and crop of the Control panel in Figure 4A of “Minocycline inhibits caspase activation and reactivation, increases the ratio of XIAP to smac/DIABLO, and reduces the mitochondrial leakage of cytochrome C and smac/DIABLO” Scarabelli etal, Journal of the American College of Cardiology, 43, 865–874 (2004) https://doi.org/10.1016/j.jacc.2003.09.050"

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  Public: Enhanced IL-17 signalling following myocardial ischaemia/reperfusion injury
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